Malaria serebral:Suatu Tinjauan Pustaka
| Main Author: | Perpustakaan UGM, i-lib |
|---|---|
| Format: | Article NonPeerReviewed |
| Terbitan: |
[Yogyakarta] : Universitas Gadjah Mada
, 1990
|
| Subjects: | |
| Online Access: |
https://repository.ugm.ac.id/24364/ http://i-lib.ugm.ac.id/jurnal/download.php?dataId=7337 |
Daftar Isi:
- ABSTRAK Cerebral malaria is one of the most serious complications of P. falaparsan infection and is defined as acute, diffuse, symetric encephalopathy in patiens with falaparum malaria. The principal cause of the pathology of cerebral malaria is due to blockage of cerebral capillaries by P. falciparum infected erythrocytes. In fatal cerebral malaria cases, the brain tissue and the cortex cerebri capillaries are destruml by infected erythrocytes. The most important clinical manifestations of cerebral malaria are fever, headache, myalgia, body weakness, mental confusion, coma and convulsion. The neurologic sequelae of cerebral malaria include hemisensory loss, hemiparesis and general seizure. The diagnosis of cerebral malaria is based on the isolation of P. fakiparum from the blood or the detection of antibodies against P. faldparum. Intravenous or parenteral quinine therapy of cerebral malaria is a priority. A regimen of quinine 10 mg or 20 mg/kgBW every 8 hours for 3 days or 4 days is administred to cerebral malaria patients and followed by oral quinine dihydrochloride. Key words : cerebral malaria, clinical manifestation, diagnosis, therapy